JUDY A. MIKOVITS, Ph.D.

Laboratory of Leukocyte Biology, Division of Basic Sciences, National Cancer Institute, Frederick Cancer Research and Development Center, National Institutes of Health

mikovits@ncifcrf.gov

Dr. Mikovits obtained her Ph.D. in Biochemistry and Molecular Biology from George Washington University. Her doctoral research focused on HIV-1 latency under the direction of Francis Ruscetti. Dr. Mikovits performed postdoctoral work on the molecular genetics of HTLV-1 under David Derse at the National Cancer Institute-FCRDC.

The mechanisms by which human retroviruses alter the function of the immune system and other host responses resulting in pathogenesis are not well understood. The current focus of our studies is to define viral and cellular factors involved in pathogenesis. Specifically, we have examined viral and cellular factors involved in regulating HIV infectivity and expression, cell death and mechanisms of immune dysfunction. In this regard, we and others have shown that HIV-1 expression in monocytes and helper T lymphocytes can be silenced and that this latency can be overcome by both immune activation and hypomethylation. Studies of SIV and HIV infection in the lymph nodes indicate that the capacity of the host to control viral load is predictive of disease progression.  The importance of latent reservoirs is underscored by recent studies showing patients in which plasma virus is reduced to undetectable levels following highly active triple drug antiretroviral therapy, however soon after cessation of therapy high viral titers are again detected, suggesting long lived reservoirs of latent virus exist.  Therefore, we have focused our efforts on examining infectivity of HIV-1 and HTLV-1 and defining cytokines and other cellular mechanisms involved in maintaining a balance between human retroviral expression and latency.

Recent Publications:

Last revised on October 26, 1998 by D. Dixon.


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